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3,721 documents 34 sections 43,623 citations 34,854 keywords indexed 4 evidence tiers

3,633 are the core, quality-scored corpus (34 lettered sections — see How We Work); the remaining 88 are cross-corpus synthesis documents (68 InterDocs, 12 Connections, 8 Theories) also indexed here.

15 results for "prion diseases"

Z_2_08 Molecular Biology

Z_2_08 — Prion Genetics and Misfolded Proteins

Prions are infectious agents composed entirely of misfolded protein — the only known pathogen that contains no nucleic acid (no DNA, no RNA). The protein-only hypothesis (Stanley Prusiner, 1982 — Nobel Prize 1997) states

prion PRNP PrP PrPSc PrPC prion diseases
Z_4_18 Verified Molecular Biology

Z_4_18 — Protein Misfolding and Prion Diseases

Prion diseases — transmissible spongiform encephalopathies (TSEs) — are fatal neurodegenerative disorders caused by the misfolding and self-propagating aggregation of a normal cellular protein (PrPᶜ) into a pathological

prion protein-misfolding amyloid bse cjd mad-cow-disease
X_5_03 Verified Medicine & Healing

X_5_03 — Medical Genetics and Rare Diseases

Medical genetics is the branch of medicine concerned with the diagnosis, management, and counseling of individuals and families affected by genetic disorders — conditions caused by mutations in DNA, ranging from single-g

medical genetics rare diseases genetic disorders inborn errors Garrod orphan diseases
Z_2_17 Verified Molecular Biology

Z_2_17 — Prion Biology: Self-Propagating Protein Misfolding and Transmissible Encephalopathies

Prions — proteinaceous infectious particles lacking nucleic acid — represent a paradigm-shattering departure from the central dogma that biological information flows from DNA to RNA to protein. The protein-only hypothesi

prion PrPSc PrPC transmissible spongiform encephalopathy Stanley Prusiner mad cow disease
Z_2_20 Verified Molecular Biology

Z_2_20 — Prion Molecular Biology

At the molecular level, prion diseases arise from the conversion of the normal cellular prion protein (PrPᶜ) into a misfolded, aggregation-prone conformer (PrPˢᶜ) through a process that remains one of the most extraordin

prion PrP protein misfolding amyloid conformational change PrPSc
R_5_17 Verified Biology & Evolution

R_5_17 — Prion Biology and Ecology

Prions — infectious agents composed entirely of misfolded protein, devoid of nucleic acid — represent one of the most conceptually revolutionary discoveries in biology, fundamentally challenging the central dogma that ge

prion PrP transmissible spongiform encephalopathy TSE mad cow disease BSE
X_4_12 Verified Medicine & Healing

X_4_12 — Tropical Medicine: Disease, Ecology, and Global Health in the Tropics

Tropical medicine is the branch of medicine concerned with diseases that are prevalent or unique to tropical and subtropical regions — particularly vector-borne diseases (malaria, dengue, yellow fever, Chagas disease, le

tropical medicine neglected tropical diseases malaria dengue Chagas schistosomiasis
Z_4_09 Verified Molecular Biology

Z_4_09 — Protein Folding: From Anfinsen's Dogma to AlphaFold

Protein folding — the process by which a linear chain of amino acids spontaneously adopts its specific three-dimensional structure — is one of the most fundamental problems in molecular biology and has been called the "s

protein folding Anfinsen AlphaFold Levinthal paradox chaperones folding funnel
Z_4_23 Verified Molecular Biology

Z_4_23 — Memory as Physical and Molecular Phenomenon

What is a memory made of? The question has driven neuroscience from Santiago Ramón y Cajal's 1894 hypothesis that learning strengthens connections between neurons, through Donald Hebb's 1949 postulate that "neurons that

molecular memory memory engram synaptic plasticity long-term potentiation LTP Eric Kandel
Z_4_16 Verified Molecular Biology

Z_4_16 — Phase Separation in Cell Biology: Membraneless Organelles and Biomolecular Condensates

Liquid-liquid phase separation (LLPS) is the biophysical process by which proteins and nucleic acids demix from the surrounding cytoplasm or nucleoplasm to form concentrated, membrane-free droplets called biomolecular co

phase separation biomolecular condensate membraneless organelle liquid-liquid phase separation LLPS intrinsically disordered protein
Verified

INTERDOC_53 — Substrate-Independent Information Patterns: Empirical Cases

A pattern is empirically substrate-independent if the same information content is preserved across changes in the physical material carrying it. Across multiple domains, biology and physics provide concrete instances of

substrate independence information theory bioelectric memory planarian regeneration prion proteins epigenetic inheritance
S_4_01 Future Technology

S_4_01 — Existential Risk Taxonomy

Existential risk (x-risk) refers to any event that could permanently curtail humanity's long-term potential — including extinction, civilizational collapse without recovery, or irreversible loss of value (e.g., permanent

existential risk x-risk global catastrophic risk GCR extinction Bostrom
I_5_09 Credible UAP Disclosure

I_5_09 — Cattle Mutilation and UAP Association

Cattle mutilation refers to the unexplained deaths of livestock — predominantly cattle — found with specific organs or tissue removed with what witnesses describe as "surgical precision," often accompanied by complete or

cattle mutilation animal mutilation surgical precision exsanguination predator exclusion UFO mutilation link
Z_2_09 Molecular Biology

Z_2_09 — Mitochondrial Genetics and Diseases

Human mitochondrial DNA (mtDNA) is a 16,569-bp circular genome encoding 37 genes: 13 proteins (all subunits of the oxidative phosphorylation/OXPHOS complexes I, III, IV, and V), 22 transfer RNAs, and 2 ribosomal RNAs. Un

mitochondrial genetics mtDNA mitochondrial DNA mitochondrial disease oxidative phosphorylation OXPHOS
L_5_02 Verified Genetics & Origins

L_5_02 — Genetic Diseases and Founder Effect Populations

When a small group founds a new population and subsequently expands in relative isolation, genetic drift can amplify alleles that were rare in the ancestral population — including deleterious recessive disease alleles. T

founder effect genetic disease Tay-Sachs sickle cell cystic fibrosis Ashkenazi